Schumacher Lab Publications

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Age is in the nucleus

Schumacher B, Vijg J
Age is in the nucleus
Nat Metab. 2019. doi: 10.1038/s42255-019-0125-9. Epub 2019 Oct 7

Age is in the nucleus

Schumacher B, Vijg J
Age is in the nucleus
Nat Metab. 2019. doi: 10.1038/s42255-019-0125-9. Epub 2019 Oct 7

Improved Biocompatibility of Amino-functionalized Graphene Oxide in Caenorhabditis elegans.

Rive C, Reina G, Wagle P, Treossi E, Palermo V, Bianco A, Gemma-Delogu L, Rieckher M#, Schumacher B#.
Improved Biocompatibility of Amino-functionalized Graphene Oxide in Caenorhabditis elegans.
Small 2019, 1902699
(#co-corresponding authors)

Recent advances in understanding the mechanisms determining longevity.

Bayersdorf R, Schumacher B.
Recent advances in understanding the mechanisms determining longevity.
F1000Res. 2019 Aug 9;8. pii: Faculty Rev-1403. doi: 10.12688/f1000research. 19610.1. eCollection 2019.

Somatic Niche Cells Regulate the CEP-1/p53-Mediated DNA Damage Response in Primordial Germ Cells

Ou HL, Kim CS, Uszkoreit S, Wickström SA, Schumacher B.
Somatic Niche Cells Regulate the CEP-1/p53-Mediated DNA Damage Response in Primordial Germ Cells
Dev Cell. 2019 Jul 22;50(2):167-183.e8. doi: 10.1016/j.devcel.2019.06.012.

ALG-2/AGO-Dependent mir-35 Family Regulates DNA Damage-Induced Apoptosis Through MPK-1/ERK MAPK Signaling Downstreamof the Core Apoptotic Machinery in Caenorhabditis elegans.

Doll MA, Soltanmohammadi N, Schumacher B.
ALG-2/AGO-Dependent mir-35 Family Regulates DNA Damage-Induced Apoptosis Through MPK-1/ERK MAPK Signaling Downstreamof the Core Apoptotic Machinery in Caenorhabditis elegans.
Genetics. 2019 Jul 11. pii: genetics.302458.2019. doi: 10.1534/genetics.119.302458.

Restorationof Proteostasis in the Endoplasmic Reticulum Reverses an Inflammation-Like Response to Cytoplasmic DNA in Caenorhabditis elegans.

Williams AB, Heider F, Messling JE, Riekher M, Bloch W, Schumacher B.
Restorationof Proteostasis in the Endoplasmic Reticulum Reverses an Inflammation-Like Response to Cytoplasmic DNA in Caenorhabditis elegans.
Genetics. 2019 Jun 27. pii: genetics.302422.2019. doi: 10.1534/genetics.119302422. [Epub ahead of print]

UBQLN4 represses homologous recombination and is overexpressed in aggressive tumors.

Jachimowicz RD, Beleggia F, Isensee J, Bhavana VB, Goergens J, Bustos MA, Doll MA, Shenoy A, Checa-Rodriguez C, Wiederstein JL, Baranes-Bachar K, Bartenhagen C, Hertwig F, Teper N, Nishi T, Schmitt A, Distelmaier F, Lüdecke HJ, Albrecht B, Krüger M, Schumacher B, Geiger T, Hoon DSB, Huertas P, Fischer M, Hucho T, Pfeifer M, Ziv Y, Reinhardt HC, Wieczorek D, Shiloh Y.
UBQLN4 represses homologous recombination and is overexpressed in aggressive tumors.
Cell, 2019 Jan 24;176, 1-15. doi: 10.1016/j.cell.2018.11.024

Extension of longevity and reduction of inflammation is ovarian-dependent, but germ cell-independent in post-reproductive female mice.

Habermehl TL, Parkinson KC, Hubbard GB, Ikeno Y, Engelmeyer JI, Schumacher B, Mason JB.
Extension of longevity and reduction of inflammation is ovarian-dependent, but germ cell-independent in post-reproductive female mice.
Geroscience. 2018 Dec 13. doi: 10.1007/s11357-018-0049-4. [Epub ahead of print]

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